Glucose Levels And Risk Of Dementia

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An examination of the relation between glucose levels and dementia.

I recently read a thought provoking article recently with some very interesting findings. It was published in the New England Journal of Medicine in 2013. It’s a little thick with medical terminology but made some very interesting discoveries. There are a lot of experts who are starting to refer to dementia (such as Alzheimer’s disease) as type 3 diabetes based on findings similar to the findings in this study. Bottom line of the article: even low levels of blood glucose elevation over time, will severely increase risk for early dementia. So those levels that most doctors consider normal, may be more dangerous than we realized.

It is the central adiposity or “belly fat” that causes insulin resistance and leads to chronically elevated glucose levels. This is one of the leading causes of heart disease, stroke and now we realize, dementia. It is very unfortunate that so many people suffer from disease that can be prevented. Although our programs lead people to have more energy and look better than they have in years, the part I like as a physician is the prevention of these deadly diseases.

Sadly though, most people do not address the root causes of their difficulties and so wind up carrying around a bunch of extra weight and suffering from insulin resistance, chronic inflammation and disease. It’s also the “belly fat” that is pro-inflammatory. Until patients address the root causes, which are very often hormone and metabolic imbalances, they will most likely never succeed in losing the weight they desperately need to lose and in fact, continue to decline.

I hope that you’re considering joining one of our programs so that we can halt these processes that may already be at work inside of you. Whenever you are ready to make the commitment to your health that will allow you to feel terrific, look great and prevent chronic disease/disability, contact us immediately.

Read the original article by clicking on this link.

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